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SOCE, mitochondria, and inflammation

Foley, John F.

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May 28, 2019

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10.1126/scisignal.aay1437

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Abstract:

Store-operated calcium entry regulates mitochondrial function to support pathogenic TH17 cells and promote inflammation. Store-operated calcium entry regulates mitochondrial function to support pathogenic TH17 cells and promote inflammation. Store-operated calcium entry regulates mitochondrial function to support pathogenic TH17 cells and promote inflammation.

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